We use scale-free networks to study properties of the infected mass of the network during a spreading process as a function of the infection probability and the structural scaling exponent . We use the standard SIR model and investigate in detail the distribution of , We find that for dense networks this function is bimodal, while for sparse networks it is a smoothly decreasing function, with the distinction between the two being a function of . We thus recover the full crossover transition from one case to the other. This has a result that on the same network a disease may die out immediately or persist for a considerable time, depending on the initial point where it was originated. Thus, we show that the disease evolution is significantly influenced by the structure of the underlying population.

生成讲解博客的中文版本

生成AI智能校验的中文版本